J Neurocrit Care.  2023 Jun;16(1):51-52. 10.18700/jnc.220084.

Posterior reversible encephalopathy syndrome superimposed on neuronal intranuclear inclusion disease

• Kim N
• Sung E
• Lee S
• Park J

• KMID: 2543394
• DOI: http://doi.org/10.18700/jnc.220084

Figure

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  Fig. 1. Brain magnetic resonance imaging scans performed 12 hours after the seizure (A-D) and after 3 weeks of treatment (E, F). (A, B) Fluid-attenuated inversion recovery (FLAIR) revealed high signal intensity lesion at the bilateral parietooccipital lobe, right temporal lobe, and cerebellum. (E, F) The follow-up image showed less prominent high FLAIR signal intensity at the cerebellum and parietooccipital lobes. (C, D) Diffusion-weighted imaging (DWI) showed bilateral symmetric diffusion restriction lesions in the corticomedullary junction, at the frontal, parietal, and temporal lobes. (G, H) The follow-up DWI showed slightly worsened diffusion restriction at the bilateral frontotemporal lobes.

Reference

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