Yonsei Med J.  2011 Nov;52(6):1022-1024. 10.3349/ymj.2011.52.6.1022.

Successful Catheter Ablation of Focal Automatic Left Ventricular Tachycardia Presented with Tachycardia-Mediated Cardiomyopathy

Affiliations
  • 1Cardiology Division, Department of Internal Medicine, Kwandong University College of Medicine, Goyang, Korea.
  • 2Cardiology Division, Department of Internal Medicine, Kyung Hee University College of Medicine, Seoul, Korea. jinbbai@khu.ac.kr
  • 3Cardiology Division, Yonsei Cardiovascular Center and Cardiovascular Research Institute, Yonsei University College of Medicine, Seoul, Korea.

Abstract

Non-reentrant focal tachycardias occur spontaneously, facilitated by catecholamine infusion, but they cannot be initiated or terminated with programmed stimulation. These tachycardias exhibit early activation before the QRS, however, do not typically show the mid-diastolic potential that is crucial for reentrant tachycardia maintenance. Electrophysiological studies are useful for distinguishing focal from macro-reentrant ventricular tachycardia. We report herein a case of patient without a history of structural heart disease who presented with a focal Purkinje ventricular tachycardia and heart failure. The focal Purkinje ventricular tachycardia was eliminated by radiofrequency catheter ablation. All of the patien's symptoms were improved after ablation.

Keyword

Ventricular tachycardia; ventricular dysfunction; catheter ablation

MeSH Terms

Adult
Cardiomyopathies/*surgery
Catheter Ablation/*methods
Humans
Male
Tachycardia, Ventricular/*surgery
Ventricular Dysfunction/surgery

Figure

  • Fig. 1 Twelve-lead ECG upon admission revealed wide QRS tachycardia with RBBB morphology. The QRS axis was -77 degrees, and atrioventricular dissociation was observed. ECG, electrocardiogram; RBBB, right bundle branch block.

  • Fig. 2 Intracardiac-electrogram during ventricular tachycardia. Black arrow indicates Purkinje potential, which was recorded 32 ms before the onset of the QRS.

  • Fig. 3 (A) Chest X-ray taken before discharge. (B) Chest X-ray at outpatient clinic six months after discharge revealed decreased cardiac size.


Reference

1. Nogami A. Idiopathic left ventricular tachycardia: assessment and treatment. Card Electrophysiol Rev. 2002. 6:448–457.
2. Latif S, Dixit S, Callans DJ. Ventricular arrhythmias in normal hearts. Cardiol Clin. 2008. 26:367–380. vi
Article
3. Markowitz SM, Lerman BB. Mechanisms of focal ventricular tachycardia in humans. Heart Rhythm. 2009. 6:S81–S85.
Article
4. Zeppenfeld K, Blom NA, Bootsma M, Schalij MJ. Incessant ventricular tachycardia in fulminant lymphocytic myocarditis: Evidence for origin in the Purkinje system and successful treatment with ablation. Heart Rhythm. 2007. 4:88–91.
5. Castro-Rodriguez J, Verbeet T, Morissens M, Lomas O, Tran-Ngoc E, Malekzadeh-Milani S, et al. Complicated Forms of Tachycardia-Mediated Cardiomyopathy Associated with Idiopathic Left Ventricular Tachycardia. Pacing Clin Electrophysiol. 2010.
Article
6. Seiler J, Lee JC, Roberts-Thomson KC, Stevenson WG. Intracardiac echocardiography guided catheter ablation of incessant ventricular tachycardia from the posterior papillary muscle causing tachycardia--mediated cardiomyopathy. Heart Rhythm. 2009. 6:389–392.
Article
Full Text Links
  • YMJ
Actions
Cited
CITED
export Copy
Close
Share
  • Twitter
  • Facebook
Similar articles
Copyright © 2024 by Korean Association of Medical Journal Editors. All rights reserved.     E-mail: koreamed@kamje.or.kr