Dement Neurocogn Disord.  2017 Jun;16(2):54-55. 10.12779/dnd.2017.16.2.54.

Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer's Disease?

Affiliations
  • 1Department of Neurology, Veterans Health Service Medical Center, Seoul, Korea. hippocam@naver.com

Abstract

No abstract available.


MeSH Terms

Alzheimer Disease*
Brain Injuries*

Figure

  • Fig. 1 Brain computed tomography (CT) showing acute intracerebral hemorrhage in both the frontal and in the left anterior temporal areas, subarachnoid hemorrhage in the both frontal convexities (A), and epidural hemorrhage in the right frontoparietal area (B). A follow up brain CT 16 days after admission, showing resolving process of the intracerebral hemorrhage, both frontal, left anterior temporal area, associated subarachnoid hemorrhage in the both frontal convexities (C), subdural hemorrhage over both the frontal convexities, and focal epidural hemorrhage in the right frontoparietal area (D). An 18F fluorodeoxyglucose positron emission tomography showing preserved metabolism of the primary sensorimotor cortex, primary visual cortex, and subcortical structures consistent with the findings of typical AD (E, F, and G). Markedly decreased metabolism in the left inferior frontal lobe and left anterior temporal lobe, congruous with the hemorrhagic lesions on the CT scan (H).


Reference

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