J Cardiovasc Imaging.  2019 Jan;27(1):70-72. 10.4250/jcvi.2019.27.e5.

Loeffler Endocarditis: A Diagnosis Made with Cardiovascular Magnetic Resonance

Affiliations
  • 1Department of Internal Medicine, Rush University Medical Center, Chicago, IL, USA. dinesh_kalra@rush.edu

Abstract

No abstract available.


MeSH Terms

Diagnosis*
Hypereosinophilic Syndrome*

Figure

  • Figure 1 Echo (apical 4 chamber) with perflutren contrast showing a 2.4 × 3.8 cm non-perfused mass at the LV apex suggestive of thrombus or infiltrative tumor. LA: left atrium, LV: left ventricle, RV: right ventricle.

  • Figure 2 (A) Cardiovascular magnetic resonance – Steady State Free Precession (SSFP) still image (apical 2 chamber) showing normal chamber sizes with increased focal apical wall thickness of 22 mm and hypokinesis of this segment. A small pericardial effusion is also seen. (B) First pass perfusion shows hypoperfused apical and lateral endocardium along with a layer of clot lining these segments. (C) Three-chamber late gadolinium enhancement shows fibrosis in the apex in the endocardium extending to the mid myocardium in the shape of a cap. The laminated hypointense clot (arrowhead) produces a “double V” sign when contrasted with the underlying hyperintense fibrosis (asterisk). (D) Three-chamber early gadolinium enhancement with long TI of 600 ms shows dark hypointense thrombus at the apex distinct from the underlying scarred myocardium. LA: left atrium, LV: left ventricle, RV: right ventricle.


Reference

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