1. Flammer J, Orgül S, Costa VP, et al. The impact of ocular blood flow in glaucoma. Pro Retin Eye Res. 2002; 21:359–93.
Article
2. Flammer J, Pache M, Resink T. Vasospasm, its role in the pathogenesis of disease with paticular reference to eye. Pro Retin Eye Res. 2001; 20:319–49.
3. Nicolela MT. Clinical clues of vascular dysregulation and its association with glaucoma. Can J Ophthalmol. 2008; 43:337–41.
Article
4. Gherghel D, Orgul S, Dubler B, et al. Is vascular regulation in the central retinal artery altered in persons with vasospasm? Arch Ophthalmol. 1999; 117:1359–62.
Article
5. Flammer J, Pache M, Resink T. Vasospasm, its role in the pathogenesis of diseases with particular reference to the eye. Prog Retin Eye Res. 2001; 20:319–49.
Article
6. Phelps CD, Corbet JJ. Migraine and low-tension glaucoma: a case-control study. Invest Ophthalmol Vis Sci. 1985; 26:1105–8.
7. Pache M, Krauchi K, Cajochen C, et al. Cold feet and prolonged sleep-onset latency in vasosapstic syndrome. Lancet. 2001; 358:125–6.
8. Teuchner B, Orgul S, Ulmer H, et al. Reduced thirst in patients with a vasospastic syndrome. Acta Ophthalmol Scand. 2004; 82:738–40.
Article
9. Orugul S, Kaiser HJ, Flammer J, Gasser P. Systemic blood pressure and capillary blood-cell velocity in glaucoma patients: a preliminary study. Eur J Ophthalmol. 1995; 5:88–91.
10. Pache M, Dubler B, Flammer J. Peripheral vasospasm and nocturnal blood pressure dipping-two distinct risk factors for glaucomatous damage? Eur J Ophthalmol. 2003; 49:164–8.
11. Chai E, Goldberg I, Chia A, et al. Visual field responses to a hand vibration stimulus. Surv Ophthalmol. 1999; 43:S79–86.
Article
12. Flammer J. Psychophysical mechanisms and treatment of vasospastic disorders in normal-tension glaucoma. Bull Soc Belge Ophthalmol. 1992; 244:129–34.
13. Wunderlich K, Zimmerman C, Gutmann H, et al. Vasospastic persons exhibit differential expression of ABC-transport proteins. Mol Vis. 2003; 9:756–61.
14. Waldmann E, Gasser P, Dubler B, et al. Silent myocardial ischemia in glaucoma and cataract patients. Graefes Arch Clin Exp Ophthalmol. 1996; 234:595–8.
Article
15. Surgiyama T, Utsunomiya K, Ota H, et al. Comparative study of cerebral blood flow in patients with normal-tension glaucoma and control subjects. Am J Opthalmol. 2006; 141:394–6.
16. Levene RZ. Low tension glaucoma: a critical review and new material. Surv Ophthalmol. 1980; 24:621–64.
Article
17. Feuer WJ, Anderson DR. Static threshold asymmetry in early glaucomatous visual field loss. Ophthalmology. 1989; 96:1285–97.
Article
18. Poinoosawmy D, Fontana L, Wu JX, et al. Frequency of asymmetric visual field defects in normal-tension and high-tension glaucoma. Ophthalmology. 1998; 105:988–91.
Article
19. Kim C, Kim TW. Comparision of risk factors for bilateral and unilateral eye involvement in normal-tension glaucoma. Invest Ophthalmol Vis Sci. 2009; 50:1215–20.
20. Cartwright MJ, Anderson DR. Correlation of asymmetric damage with asymmetric intraocular pressure in normal-tension glaucoma (low-tension glaucoma). Arch Ophthalmol. 1988; 106:898–900.
Article
21. Crichton A, Drance SM, Douglas GR, Schulzer M. Unequal intraocular pressure and its relation to asymmetric visual field defects in low-tension glaucoma. Ophthalmology. 1989; 96:1312–4.
Article
22. Park HJ, Choi BG. The quantitative evaluation of optic nerve head of normal tension glaucoma. J Korean Ophthalmol Soc. 2000; 41:1203–9.
23. Klein BE, Klein R, Linton KL. Intraocular pressure in an American community. The Beaver Dam Eye Study. Invest Ophthalmol Vis Sci. 1992; 33:2224–8.
24. Jonas JB, Berenshtein E, Holbach L. Lamina cribrosa thickness and spatial relationships between intraocular space and cerebrospinal fluid space in highly myopic eyes. Invest Ophthalmol Vis Sci. 2004; 45:2660–5.
Article
25. Collaborative Normal-Tension Glaucoma Study Group. Comparison of glaucomatous progression between untreated patients with normal-tension glaucoma and patients with therapeutically reduced intraocular pressure. Am J Ophthalmol. 1998; 126:487–97.
26. Drance S, Anderson DR, Schulzer M. Risk factors for progression of visual field abnormalities in normal-tension glaucoma. Am J Ophthalmol. 2001; 131:699–708.
Article
27. Kitazawa Y, Shirato S, Yamamoto T. Optic disc hemorrhage in low-tension glaucoma. Ophthalmology. 1986; 93:853–7.
Article
28. Lee DH, Kee C. abdominal, abdominal comparision in normal tension glaucoma with unilateral field loss. J Korean Ophthalmol Soc. 2008; 49:125–34.
29. Jonas JB, Papastathopoulos KI. Pressure-dependent changes of the optic disk in primary open-angle glaucoma. Am J Ophthalmol. 1995; 119:313–7.
Article
30. Park KH, Tomita G, Liou SY, Kitazawa Y. Correlation between abdominal atrophy and optic nerve damage in normal-tension glaucoma. Ophthalmology. 1996; 103:1899–906.
31. Burk RO, Rohrschneider K, Noack H, Volcker HE. Are large optic nerve heads susceptible to glaucomatous damage at normal intraocular pressure? A three-dimensional study by laser scanning tomography. Graefes Arch Clin Exp Ophthalmol. 1992; 230:552–60.
32. Tuulonen A, Airaksinen PJ. Optic disc size in exfoliative primary open angle, and low-tension glaucoma. Arch Ophthalmol. 1992; 110:211–3.
Article
33. Yim JH, Park SC, Kee CW. Evaluation of ocular risk factors related to asymmetric visual field defects in normal tension glaucoma. J Korean Ophthalmol Soc. 2008; 49:1507–14.
Article