- Water and Sodium Regulation in Heart Failure
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Bae EH, Ma SK
- KMID: 2052317
- Electrolyte Blood Press.
- 2009 Dec;7(2):38-41.
- doi: 10.5049/EBP.2009.7.2.38
Heart failure is the pathophysiological state characterized by ventricular dysfunction and associated clinical symptoms. Decreased cardiac output or peripheral vascular resistance lead to arterial underfilling. That is an important signal... -
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- Altered Regulation of Renal Sodium Transporters in Salt-Sensitive Hypertensive Rats Induced by Uninephrectomy
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Jung JY, Lee JW, Kim S, Jung ES, Jang HR, Han JS, Joo KW
- KMID: 2052320
- Electrolyte Blood Press.
- 2009 Dec;7(2):58-66.
- doi: 10.5049/EBP.2009.7.2.58
Uninephrectomy (uNx) in young rats causes salt-sensitive hypertension (SSH). Alterations of sodium handling in residual nephrons may play a role in the pathogenesis. Therefore, we evaluated the adaptive alterations of... -
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- A Case of Gitelman's Syndrome Presented with Chest Pain and Syncope
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Shin HS, Park CH
- KMID: 1867705
- Korean J Nephrol.
- 2006 Nov;25(6):1013-1017.
Gitelman's syndrome is a rare autosomal recessive, inherited renal tubular disorder, first described by Gitelman et al. in 1966, and it is characterized by hypokalemic metabolic alkalosis, hypomagnesemia, salt wasting,... -
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- A Case of Gitelman Syndrome
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Han YJ, Yeon EK, Kim YC
- KMID: 2317667
- Soonchunhyang Med Sci.
- 2012 Dec;18(2):145-147.
Gitelman's syndrome is an autosomal recessive disorder characterized by hypokalemic metabolic alkalosis, hypomagnesemia, and hypocalciuria that has recently been reported to be linked to thiazide-sensitive Na-Cl cotransporter gene mutation. We... -
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- Ca2+ is a Regulator of the WNK/OSR1/NKCC Pathway in a Human Salivary Gland Cell Line
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Park S, Ku SK, Ji HW, Choi JH, Shin DM
- KMID: 1791426
- Korean J Physiol Pharmacol.
- 2015 May;19(3):249-255.
- doi: 10.4196/kjpp.2015.19.3.249
Wnk kinase maintains cell volume, regulating various transporters such as sodium-chloride cotransporter, potassium-chloride cotransporter, and sodium-potassium-chloride cotransporter 1 (NKCC1) through the phosphorylation of oxidative stress responsive kinase 1 (OSR1) and... -
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- Gitelman syndrome combined with complete growth hormone deficiency
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Min SR, Cho HS, Hong J, Cheong HI, Ahn SY
- KMID: 1429681
- Ann Pediatr Endocrinol Metab.
- 2013 Mar;18(1):36-39.
- doi: 10.6065/apem.2013.18.1.36
Gitelman syndrome is a rare autosomal recessive hereditary salt-losing tubulopathy, that manifests as hypokalemic metabolic alkalosis, hypomagnesemia, and hypocalciuria. It is caused by mutations in the solute carrier family 12(sodium/chloride... -
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- Expression of Occludin in Porcine Renal Epithelial Cells
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Lee SY, Han KH
- KMID: 1472503
- Korean J Anat.
- 2009 Mar;42(1):11-17.
Occludin is a cell adhesion molecule that is abundantly expressed in the kidney. However, the expression pattern in various renal epithelial cells is not well established. The purpose of this... -
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- Increased Expression of Sodium Transporters in Rats Chronically Inhibited of Nitric Oxide Synthesis
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Kim JS, Choi KC, Jeong MH, Kim SW, Oh YW, Lee JU
- KMID: 2157772
- J Korean Med Sci.
- 2006 Feb;21(1):1-4.
- doi: 10.3346/jkms.2006.21.1.1
The present study was done to determine whether endogenous nitric oxide (NO) plays a role in the regulation of sodium transporters in the kidney. Male Sprague-Dawley rats were treated with... -
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- Attenuated Renal Excretion in Response to Thiazide Diuretics in Gitelman's Syndrome: A Case Report
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Yeum CH, Kim SW, Ma SK, Ko JH, Nah MY, Kim NH, Choi KC
- KMID: 1109772
- J Korean Med Sci.
- 2002 Aug;17(4):567-570.
- doi: 10.3346/jkms.2002.17.4.567
Gitelman's syndrome is a variant of Bartter's syndrome characterized by hypocalciuria and hypomagnesemia. The administration of thiazide diuretics may induce a subnormal increase of urinary Na+ and Cl- excretion in... -
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- Absence of intact thiazide-sensitive sodium-chloride cotransporter in the renal tissue of a Gitelman's syndrome patient
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Jang HR, Heo NJ, Son MJ, Lee JW, Lee JH, Jeon US, Shin SJ, Na KY, Joo KW, Lee JS, Cheong HI, Kim J, Han JS
- KMID: 2081172
- Korean J Med.
- 2005 Dec;69(6):642-650.
BACKGROUND: Gitelman's syndrome is an autosomal recessive renal tubular disorder characterized by hypokalemic metabolic alkalosis, hypomagnesemia, and hypocalciuria. It is known to be caused by a mutation of SLC12A3 gene... -
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