Dement Neurocogn Disord.  2017 Dec;16(4):132-133. 10.12779/dnd.2017.16.4.132.

Transient Global Amnesia Caused by Bilateral Medial Temporal-Lobe Infarction

Affiliations
  • 1Department of Neurology, Veterans Health Service Medical Center, Seoul, Korea. hippocam@naver.com

Abstract

No abstract available.


MeSH Terms

Amnesia, Transient Global*
Infarction*

Figure

  • Fig. 1 Brain images. DWI (A) after 12 h from symptom onset reveals a small lesion of high signal intensity in the left cerebellum (arrow) with an ADC map showing the corresponding lesion (B). The arrow indicates changes in the signal intensity. DWI (C, D) shows high signal intensity in right medial temporal lobe (arrow) and the bilateral medial temporal lobe, inside the marked squares. Magnified DWI and ADC of the bilateral hippocampus lesion marked with squares in figure (D) show 3.5 mm-sized round signal changes in the right hippocampus (E, F) and 8.6 mm-sized ovoid signal changes in the left hippocampus (G, H). ADC: apparent diffusion coefficient, DWI: diffusion-weighted imaging.


Reference

1. Bartsch T, Deuschl G. Transient global amnesia: functional anatomy and clinical implications. Lancet Neurol. 2010; 9:205–214.
Article
2. Yang Y, Kim S, Kim JH. Ischemic evidence of transient global amnesia: location of the lesion in the hippocampus. J Clin Neurol. 2008; 4:59–66.
Article
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