Ann Lab Med.  2012 Nov;32(6):450-451.

Hemoglobin Andrew-Minneapolis: Hemolytic Erythrocytosis and Severe Iron Overload in Toxic Liver Cirrhosis

Affiliations
  • 1Department of Clinical Chemistry and Clinical Pharmacology, The University of Bonn, Bonn, Germany. berndt.zur@ukb.uni-bonn.de

Abstract

No abstract available.


MeSH Terms

Chromatography, High Pressure Liquid
Electrophoresis, Capillary
Hemoglobin A, Glycosylated/analysis
Hemoglobins, Abnormal/*analysis/genetics
Hemolysis
Humans
Iron Overload/complications/*diagnosis
Liver Cirrhosis/complications/*pathology
Male
Middle Aged
Polycythemia/complications/*diagnosis
Hemoglobin A, Glycosylated
Hemoglobins, Abnormal

Reference

1. Zak SJ, Brimhall B, Jones RT, Kaplan ME. Hemoglobin Andrew-Minneapolis alpha 2 A beta 2 144 Lys leads to ASN: a new high-oxygen-affinity human hemoglobin. Blood. 1974. 44:543–549.
2. Ahmed A, Jahan M, Braunitzer G, Edelbluth C, Herold W. Hb Andrew-Minneapolis [β144(HC1)Lys→ASN] in a German family from Berlin. Hemoglobin. 1989. 13:189–192.
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3. Gomi T, Ikeda T, Harano T. Hemoglobin Andrew-Minneapolis (β144 (HCI) Lysine→Asparagine) in a Japanese Family. Intern Med. 1992. 31:659–661.
4. Bonkovsky HL, Banner BF, Lambrecht RW, Rubin RB. Iron in liver diseases other than hemochromatosis. Semin Liver Dis. 1996. 16:65–82.
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5. Grifoni V, Kamuzora H, Lehmann H, Charlesworth D. A new Hb variant: Hb F Sardinia gamma 75(E19) isoleucine leads to threonine found in a family with Hb G Philadelphia, beta-chain deficiency and a Lepore-like haemoglobin indistinguishable from Hb A2. Acta Haematol. 1975. 53:347–355.
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