Endocrinol Metab.  2013 Mar;28(1):78-79. 10.3803/EnM.2013.28.1.78.

Response: The Association between Serum Endogenous Secretory Receptor for Advanced Glycation End Products and Vertebral Fractures in Type 2 Diabetes (Endocrinol Metab 2012;27:289-94, Cheol Ho Lee et al.)

• Lee CH
• Lee MK
• Han HJ
• Kim TH
• Lee JH
• Kim SH

• KMID: 1805952
• DOI: http://doi.org/10.3803/EnM.2013.28.1.78

Abstract

No abstract available.

MeSH Terms

Glycosylation End Products, Advanced
Glycosylation End Products, Advanced

Reference

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2. Yamamoto M, Yamaguchi T, Yamauchi M, Kaji H, Sugimoto T. Diabetic patients have an increased risk of vertebral fractures independent of BMD or diabetic complications. J Bone Miner Res. 2009. 24:702–709.

3. Saito M, Fujii K, Mori Y, Marumo K. Role of collagen enzymatic and glycation induced cross-links as a determinant of bone quality in spontaneously diabetic WBN/Kob rats. Osteoporos Int. 2006. 17:1514–1523.

4. Ogawa N, Yamaguchi T, Yano S, Yamauchi M, Yamamoto M, Sugimoto T. The combination of high glucose and advanced glycation end-products (AGEs) inhibits the mineralization of osteoblastic MC3T3-E1 cells through glucose-induced increase in the receptor for AGEs. Horm Metab Res. 2007. 39:871–875.