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Treatment of hypothyroidsm

Jo YS, Shong M

  • KMID: 1984654
  • Korean J Med.
  • 2005 Jul;69(1):119-120.
No abstract available.
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Identification of Growth Regulatory Factors in Medullary Thyroid Carcinoma Cell Line

Jo YS, Shong M

No abstract available.
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Epigenetic Regulation of RUNX3 in Thyroid Carcinoma

Joung KH, Shong M

  • KMID: 2432929
  • Korean J Intern Med.
  • 2012 Dec;27(4):391-393.
No abstract available.
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Postoperative Findings of the Cytological Diagnosis of Follicular Neoplasm or Hurthle Cell Neoplasm and Risk of Malignancy

Lee JU, Shong M

No abstract available.
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Pathogenetic Mechanisms and Therapeutic Implications of BRAFV600E in Papillary Thyroid Cancer

Shong M, Jo YS, Ro HK

No abstract available.
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Response: GDF15 Is a Novel Biomarker for Impaired Fasting Glucose (Diabetes Metab J 2014;38:472-9)

Hong JH, Ku BJ, Shong M

No abstract available.
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Follicular and Hurthle cell carcinoma of the thyroid in iodine-sufficient area: retrospective analysis of Korean multicenter data

Kim WG, Kim TY, Kim TH, Jang HW, Jo YS, Park YJ, Kim SW, Kim WB, Shong M, Park DJ, Chung JH, Shong YK, Cho BY

BACKGROUND/AIMS: Follicular thyroid carcinoma (FTC) and Hurthle cell carcinoma (HCC) of the thyroid are relatively uncommon thyroid malignancies in iodine-sufficient areas. In this study we evaluated the clinical behavior, prognostic...
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A Case of Multiple Endocrine Neoplasia Type I with Atypical Clinical Course

Choi YS, Bai YS, Ku BJ, Jo YS, Kim YK, Ro HK, Shong M

  • KMID: 2333264
  • J Korean Endocr Soc.
  • 2008 Aug;23(4):266-271.
Multiple endocrine neoplasia type 1 (MEN 1) is characterized by the combined occurrence of primary hyperparathyroidism, enteropancreatic tumors and anterior pituitary adenoma. Yet carcinoid tumors, adrenal adenoma and lipoma might...
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Two Cases of Simple Virilizing Congenital Adrenal Hyperplasia with Compound Heterozygous Mutations of CYP21 Gene

Kim KS, Choi YS, Bai YS, Rha SY, Jo YS, Shong M

Steroid 21-hydroxylase deficiency is the most frequent cause of congenital adrenal hyperplasia (CAH), which is an inherited inability to synthesize cortisol. Actually, CAH is caused by mutations in the CYP21...
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The Role of Growth Differentiation Factor 15 in Energy Metabolism

Chang JY, Hong HJ, Kang SG, Kim JT, Zhang BY, Shong M

Growth differentiation factor 15 (GDF15) is receiving great interest beyond its role as an aging and disease-related biomarker. Recent discovery of its receptor, glial cell line-derived neurotrophic factor (GDNF) family...
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Protocol for a Korean Multicenter Prospective Cohort Study of Active Surveillance or Surgery (KoMPASS) in Papillary Thyroid Microcarcinoma

Jeon MJ, Kang YE, Moon JH, Lim DJ, Lee CY, Lee YS, Kim SW, Kim MH, Kim BH, Kang HC, Shong M, Cho SW, Kim WB

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Development of Metabolic Synthetic Lethality and Its Implications for Thyroid Cancer

Ju SH, Lee SE, Kang YE, Shong M

Cancer therapies targeting genetic alterations are a topic of great interest in the field of thyroid cancer, which frequently harbors mutations in the RAS, RAF, and RET genes. Unfortunately, U.S....
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Oncogenes, mitochondrial metabolism, and quality control in differentiated thyroid cancer

Yi HS, Chang JY, Kim KS, Shong M

Thyroid cancer is one of the most common malignancies of endocrine organs, and its incidence rate has increased steadily over the past several decades. Most differentiated thyroid tumors derived from...
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Metabolic Reprogramming in Thyroid Cancer

Ju SH, Song M, Lim JY, Kang YE, Yi HS, Shong M

Thyroid cancer is a common endocrine malignancy with increasing incidence globally. Although most cases can be treated effectively, some cases are more aggressive and have a higher risk of mortality....
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Cellular and Intercellular Homeostasis in Adipose Tissue with Mitochondria-Specific Stress

Choi MJ, Jung SB, Chang JY, Shong M

Paracrine interactions are imperative for the maintenance of adipose tissue intercellular homeostasis, and intracellular organelle dysfunction results in local and systemic alterations in metabolic homeostasis. It is currently accepted that...
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Mitochondrial Energy Metabolism and Thyroid Cancers

Lee J, Chang JY, Kang YE, Yi S, Lee MH, Joung KH, Kim KS, Shong M

Primary thyroid cancers including papillary, follicular, poorly differentiated, and anaplastic carcinomas show substantial differences in biological and clinical behaviors. Even in the same pathological type, there is wide variability in...
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Regulation of Systemic Glucose Homeostasis by T Helper Type 2 Cytokines

Kang YE, Kim HJ, Shong M

Obesity results in an inflammatory microenvironment in adipose tissue, leading to the deterioration of tissue protective mechanisms. Although recent studies suggested the importance of type 2 immunity in an anti-inflammatory...
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Expression of miRNA 146a/b, 221 and 222 in Thyroid Cancer

Jo YS, Lee IS, Hong W, Song IS, Shong M, Kim JR

BACKGROUND: miRNAs can be diagnostic markers and therapeutic targets in cancers, but few studies have been conducted in thyroid cancer. We investigated the expression levels of miRNA 146a/b, 221, and...
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Mitochondrial Stress and Mitokines: Therapeutic Perspectives for the Treatment of Metabolic Diseases

Zhang B, Chang JY, Lee MH, Ju SH, Yi HS, Shong M

Mitochondrial stress and the dysregulated mitochondrial unfolded protein response (UPRmt) are linked to various diseases, including metabolic disorders, neurodegenerative diseases, and cancer. Mitokines, signaling molecules released by mitochondrial stress response...
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Association between DIO2 Thr92Ala polymorphism and hypertension in patients with hypothyroidism: Korean Genome and Epidemiology Study

Kang YM, Koo BS, Yi HS, Kim JT, Park B, Lee JH, Shong M, Kang YE

Background/Aims: Recent evidence has identified the significance of type 2 iodothyronine deiodinase (DIO2) in various diseases. However, the role of DIO2 polymorphism in metabolic parameters in patients with hypothyroidism is...
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