J Korean Med Sci.  1998 Oct;13(5):566-568. 10.3346/jkms.1998.13.5.566.

Superior sagittal sinus thrombosis and transient ischemic attacks: possible mechanism

• Chang DI
• Yoon SS
• Chung KC

• KMID: 645223
• DOI: http://doi.org/10.3346/jkms.1998.13.5.566

Abstract

Clinical manifestations of superior sagittal sinus (SSS) thrombosis are nonspecific but characterized by headache, papilledema, seizures, focal deficits, progressive coma and death. Recurrent transient focal neurologic deficit is an extremely rare manifestation in superior sagittal sinus thrombosis and the mechanism is unknown. A 45-year-old man presented with headache for two weeks and four episodes of transient (5-10 minutes) right or left hemiparesis for two days. Magnetic resonance image and transfemoral cerebral angiography revealed superior sagittal sinus thrombosis with numerous prominent collateral venous channels. There was no parenchymal lesion. After four days of heparinization, no further transient focal neurologic deficits developed. Follow-up angiography showed partial recanalization of the SSS. Possible mechanism of transient ischemic attacks in this patient is thought to be a transient functional disturbance due to a temporal reduction of tissue perfusion in the process of operating fully-enough collateral channels.

MeSH Terms

Case Report
Cerebral Angiography
Follow-Up Studies
Human
Ischemic Attack, Transient/therapy
Ischemic Attack, Transient/radiography
Ischemic Attack, Transient/physiopathology*
Ischemic Attack, Transient/pathology
Magnetic Resonance Imaging
Male
Middle Age
Sinus Thrombosis, Intracranial/therapy
Sinus Thrombosis, Intracranial/radiography
Sinus Thrombosis, Intracranial/physiopathology*
Sinus Thrombosis, Intracranial/pathology