J Neurocrit Care.  2021 Dec;14(2):121-122. 10.18700/jnc.210027.

Posterior reversible encephalopathy syndrome as delayed neurological sequelae after carbon monoxide intoxication

Affiliations
  • 1Department of Neurology, Hanyang University College of Medicine, Seoul, Korea


Figure

  • Fig. 1. Brain magnetic resonance imaging. (A) The day of carbon monoxide (CO) poisoning, fluid-attenuated inversion recovery (FLAIR) images showed no abnormal high signal intensity. (B) Twelve days from CO intoxication, FLAIR images showed high signal intensities in bilateral globus pallidus (red box) and both parieto-occipital cortex (yellow arrows), suggesting posterior reversible encephalopathy syndrome. (C) Twenty-five days after CO intoxication, follow-up FLAIR image was obtained from the outpatient visit, and it showed interval decrease of signal and volume in bilateral globus pallidus (green box) and temporo-parieto-occipital cortex (blue arrows).


Reference

1. Varrassi M, Di Sibio A, Gianneramo C, Perri M, Saltelli G, Splendiani A, et al. Advanced neuroimaging of carbon monoxide poisoning. Neuroradiol J. 2017; 30:461–9.
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3. Park EJ, Min YG, Jung YS, Lee S, Choi SC. Posterior reversible leukoencephalopathy syndrome following acute carbon monoxide poisoning: a case report. J Korean Soc Emerg Med. 2014; 25:120–3.
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