01). Slucky AV. Acute spinal cord injuries: Pathophysiologic mechanisms, experimental therapy, and recovery of function. In: Clark CR (ed): The Cervical Spine,. 3rded.Philadelphia: Lippincott-Raven;p. 521. 1997.
02). Fehlings MG., Ghassan SN. A review of the pathophysiology of cervical spondylotic myelopathy with insights for potential novel mechanisms drawn for traumatic spinal cord injury. Spine. 1998. 23:2730–2737.
03). Yang JY., Lee JK., Kim HS. The Changes of Apoptosis accroding to the time of Administration of Methylprednisolone after Spinal Cord Injury in Rats. The Spine Journal. 2006. 6:23.
04). Singer JM, Russel GV, Coe Je. Changes in evoked potentials after experimental cervical spinal cord injury in the monkey. Exp Neurol. 1970. 29:449–461.
05). Chao J., Xu J., Hsu CY. Kininogen and kinins in experimental spinal cord injury [abstract]. FASEB J. 1988. 2:145.
06). Delamarter RB., Sherman J., Carr JB. Pathophysiology of spinal cord injury. J Bone Joint Surg, Am. 1995. 77:1042–1049.
07). Tarlov IM., Klinger H. Spinal cord compression studies. II. Time limits for recovery after acute compression in dogs. Arch Neurol Psych. 1954. 71:271–290.
08). Vaccaro AR., Daugherty RJ., Sheenan TP, et al. Neurologic outcome of early versus late surgery for cervical spinal cord injury. Spine. 1997. 22:2609–2613.
Article
09). Vyklicky L,Sykova E. The effects of increased extracellular potassium in the isolated spinal cord on the flexor reflex of the frog. Neurosci Lett. 1975. 19:203–207.
10). Young W., Koreh I., Yen V., Lindsay A. Effect of sympathectomy on extracellular potassium activity and blood flow in experimental spinal cord contusion. Brain Res. 1982. 253:115–124.
11). Tator CH. Biology of neurologic recovery and functional restoration after spinal cord injury. Neurosurgery. 1998. 42:696–707.
12). Taoka Y., Okajima K. Spinal cord injury in the rat. Progr Neurobiol. 1998. 56:341–358.
Article
13). Senter HJ., Venes JL. Loss of autoregulation and posttraumatic ischemia following experimental spinal cord trauma. J Neurosurg. 1979. 50:198–206.
Article
14). Allen AR. Surgery of experimental lesion of spinal cord equivalent to crush injury of fracture dislocation. JAMA. 1911. 50:941–952.
15). Tator CH., Koyanagi I. Vascular mechanisms in the pathophysiology of human spinal cord injury. J Neurosurg. 1997. 86:483–492.
Article
16). Blight AR., Young W. Central axons in injured cat spinal cord recover electrophysiologic function following remyelination by Schwann cells. J Neurol Sci. 1989. 91:15–34.
17). Holaday JW., Faden AI. Naloxone acts at central opiate receptors to reverse hypotension, hypothermia and hypoventilation in spinal shock. Brain Res. 1980. 189:295–299.
Article
18). Bracken MB., Shepard MJ., Collins WF, et al. Methyl-prednisolone or naloxone treatment after acute spinal cord injury: One-year follow-up data. Results of the second NASCIS. J Neurosurg. 1992. 76:23–31.
19). Tymianski M., Tator CH. Normal and abnormal calcium homeostasis inneurons: A basis for the pathophysiology of traumatic and ischemic central nervous system injury. Neurosurgery. 1996. 38:1176–1195.
20). Balentine JD., Dean D. Calcium-induced spongiform and necritizing myelopathy. Lab Invest. 1982. 47:286–295.
21). Agrawal SK., Fehlings MG. Role of NMDA and non-NMDA ionotropic glutamate receptors in traumatic spinal cord axonal injury. J Neurosurg. 1997. 17:1055–1063.
Article
22). Faden AI., Simon RP. A potential role for excitotoxins in the pathophysiology of spinal cord injury. Ann Neurol. 1988. 23:623–626.
Article
23). Imaizumi T., Kocsis JD., Waxman SG. The role of voltage-gated Ca2+ channel sinanoxic injury of spinal cord white matter. Brain Res. 1999. 817:84–92.
24). Bethea JR., Castro M., Keane RW, et al. Traumatic spinal cord injury induces nuclear factor-kB activation. J Neurosci. 1998. 18:3251–3260.
25). Emery E., Aldana P., Bunge MB, et al. Apoptosis after traumatic human spinal cord injury. J Neurosurg. 1998. 89:911–920.
Article
26). Lenke J., Lenke LG., Ludwig FJ., O'Brien MF. Apoptosis as a mechanism of neuronal cell death following acute experimental spinal cord injury. Spinal Cord. 1998. 36:683–690.
Article
27). Bonfoco E., Kraine D., Ankarcrona M, et al. Apoptosis and necrosis: Two distinct events induced, respectively, by mild and intense insults with N-methyl-D-aspartate or nitric oxide/superoxide in cortical cell cultures. Proc Natl Acad Sci USA. 1995. 92:7162–7166.
Article
28). Dimitrijevic MR., Frangel J., Lehmkuhl D., Sherwood A. Motor control in man after partial or complete spinal cord injury. Adv Neurol. 1983. 39:915–926.