- Notch Intracellular Domain Expression in Various Skin Fibroproliferative Diseases
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Kim JE, Lee JH, Jeong KH, Kim GM, Kang H
- KMID: 2368153
- Ann Dermatol.
- 2014 Jun;26(3):332-337.
BACKGROUND: The effects of the Notch signaling pathway in fibroproliferative skin diseases have not been fully elucidated. OBJECTIVE: The aim of this study was to investigate the expression of activated Notch... -
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- Bacoside A Induced Sub-G0 Arrest and Early Apoptosis in Human Glioblastoma Cell Line U-87 MG through Notch Signaling Pathway
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Aithal MG, Rajeswari
- KMID: 2444792
- Brain Tumor Res Treat.
- 2019 Apr;7(1):25-32.
- doi: 10.14791/btrt.2019.7.e21
BACKGROUND: Glioblastoma multiforme (GBM) is a highly malignant brain tumor with a worst prognosis of less than one year despite advance treatment facilities. Among various signaling pathway genes displaying genetic... -
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- Nerve Growth Factor Stimulates Glioblastoma Proliferation through Notch1 Receptor Signaling
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Park JC, Chang IB, Ahn JH, Kim JH, Song JH, Moon SM, Park YH
- KMID: 2417319
- J Korean Neurosurg Soc.
- 2018 Jul;61(4):441-449.
- doi: 10.3340/jkns.2017.0219
OBJECTIVE: Notch receptors are heterodimeric transmembrane proteins that regulate cell fate, such as differentiation, proliferation, and apoptosis. Dysregulated Notch pathway signaling has been observed in glioblastomas, as well as in... -
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- Brain Tumor Stem Cells as Therapeutic Targets in Models of Glioma
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Laks DR, Visnyei K, Kornblum H
- KMID: 1071412
- Yonsei Med J.
- 2010 Sep;51(5):633-640.
- doi: 10.3349/ymj.2010.51.5.633
At this time, brain tumor stem cells remain a controversial hypothesis while malignant brain tumors continue to present a dire prognosis of severe morbidity and mortality. Yet, brain tumor stem... -
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- Microarray for Genes Associated with Signal Transduction in Diabetic OLETF Keratocytes
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Lee JE, Lee JS, Hwang SH
- KMID: 1101914
- Korean J Ophthalmol.
- 2007 Jun;21(2):111-119.
- doi: 10.3341/kjo.2007.21.2.111
PURPOSE: The purpose of this study was to identify differences in signal transduction gene expression between normal and diabetic keratocytes stimulated with interleukin-1alpha (IL-1alpha) and tumor necrosis factor-alpha (TNF-alpha). ... -
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