- Dysfunctional Mitochondrial Bioenergetics and Synaptic Degeneration in Alzheimer Disease
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Tang J, Oliveros A, Jang MH
- KMID: 2448802
- Int Neurourol J.
- 2019 Feb;23(Suppl 1):S5-S10.
- doi: 10.5213/inj.1938036.018
Synapses are sites of high energy demand which are dependent on high levels of mitochondrial derived adenosine triphosphate. Mitochondria within synaptic structures are key for maintenance of functional neurotransmission and... -
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- Genetic Inhibition of sFRP3 Prevents Glial Reactivity in a Mouse Model of Accelerated Aging
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Corujo-Ramirez AM, Dua M, Yoo KH, Oliveros A, Jang MH
- KMID: 2510831
- Int Neurourol J.
- 2020 Nov;24(Suppl 2):72-78.
- doi: 10.5213/inj.2040436.218
Purpose: Aging is the most significant risk factor for neurodegenerative disorders that are typified by cognitive deficits. Our recent work utilizing BubR1 hypomorphic (BubR1H/H) mice, an accelerated aging model, has... -
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- BubR1 Insufficiency Impairs Affective Behavior and Memory Function in Mice
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Cho CH, Yang Z, Yoo KH, Oliveros A, Jang MH
- KMID: 2426690
- Int Neurourol J.
- 2018 Oct;22(Suppl 3):S122-S130.
- doi: 10.5213/inj.1836218.109
PURPOSE: Although aging causes functional declines in cognition, the molecular mechanism underlying these declines remains largely unknown. Recently, the spindle checkpoint kinase budding uninhibited by benzimidazole-related 1 (BubR1) has emerged... -
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