- Islet Transplantation and Regeneration for Treatment of Diabetes
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Yamada Y
- KMID: 1726146
- Yonsei Med J.
- 2004 Jun;45(Suppl):S53-S55.
- doi: 10.3349/ymj.2004.45.Suppl.53
Islet transplantation has the potential to restore normoglycemia and prevent the development of diabetic complications such as retinopathy, nephropathy and neuropathy, and could therefore ve a valuable treatment for diabetic... -
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- Bilirubin protects grafts against nonspecific inflammation-induced injury in syngeneic intraportal islet transplantation
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Zhu H, Wang J, Jiang H, Ma Y, Pan S, Reddy S, Sun X
- KMID: 994397
- Exp Mol Med.
- 2010 Nov;42(11):739-748.
- doi: 10.3858/emm.2010.42.11.075
Nonspecific inflammatory response is the major cause for failure of islet grafts at the early phase of intraportal islet transplantation (IPIT). Bilirubin, a natural product of heme catabolism, has displayed... -
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- Prolonged survival of islet allografts in mice treated with rosmarinic acid and anti-CD154 antibody
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Jung DY, Kim EY, Joo SY, Park JB, Moon C, Kim SH, Sim EY, Joh JW, Kwon CH, Kwon GY, Kim SJ
- KMID: 1103582
- Exp Mol Med.
- 2008 Feb;40(1):1-10.
- doi: 10.3858/emm.2008.40.1.1
Pancreatic islet transplantation can correct the abnormal glucose metabolism of Type 1 diabetes. Although immunosuppressants greatly reduce the acute rejection rate in transplant patients, the long-term side effects can be... -
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- Effects of BCG, lymphotoxin and bee venom on insulitis and development of IDDM in non-obese diabetic mice
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Kim JY, Cho SH, Kim YW, Jang EC, Park SY, Kim EJ, Lee SK
- KMID: 646041
- J Korean Med Sci.
- 1999 Dec;14(6):648-652.
- doi: 10.3346/jkms.1999.14.6.648
To investigate whether BCG, lymphtoxin (LT) or bee venom (BV) can prevent insulitis and development of diabetes in non-obese diabetic (NOD) mice, we measured the degree of insulitis and incidence... -
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- Reduced dihydroxyacetone sensitivity and normal sensitivity to glyceraldehyde and oxidizing agent of ATP-sensitive K+ channels of pancreatic beta cells in NIDDM rats
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Song DK, Park WK, Bae JH, Park MK, Kim SJ, Ho WK, Earm YE
- KMID: 1081048
- J Korean Med Sci.
- 1997 Aug;12(4):286-292.
- doi: 10.3346/jkms.1997.12.4.286
The inhibition of ATP-sensitive K+(KATP) channels in pancreatic beta cells is a key step of insulin secretion induced by glucose. Glucose-induced insulin secretion from the beta cells is selectively... -
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