- Experiences of Nurses Working in a Single-Room-Structured Intensive Care Unit
-
Youn JH, Shin YM, Shin SJ, Hong EM
- KMID: 2521665
- J Korean Crit Care Nurs.
- 2021 Oct;14(3):1-13.
- doi: 10.34250/jkccn.2021.14.3.1
Purpose : This study aims to provide basic data for effective nursing interventions and improvement of nurses’ work by exploring their work experiences in single-room-structured intensive care units (ICU) through... -
- CITED
-
- Close
- SHARE
-
- Close
- Atorvastatin Induces FXR and CYP7A1 Activation as a Result of the Sequential Action of PPARγ/PGC-1α/HNF-4α in Hep3B Cells
-
Lee J, Hong EM, Jung JH, Park SW, Lee SP, Koh DH, Jang HJ, Kae SH
- KMID: 2514088
- Korean J Gastroenterol.
- 2021 Mar;77(3):123-131.
- doi: 10.4166/kjg.2020.156
Background/Aims: PPARγ, farnesoid X receptor (FXR) and CYP7A1 are associated with solubility of bile. This study was performed to understand a mechanism and interactions of statin-induced PPARγ, PGC-1α and HNF-4α... -
- CITED
-
- Close
- SHARE
-
- Close
- Ursodeoxycholic Acid Suppresses Epithelial-Mesenchymal Transition via Inhibition of Reactive Oxygen Species in Bile Duct Cancer Cells
-
Lee J, Hong EM, Lee KJ, Park SW, Koh DH
- KMID: 2551376
- Korean J Pancreas Biliary Tract.
- 2024 Jan;29(1):9-19.
- doi: 10.15279/kpba.2024.29.1.9
Background/Aim: Reactive oxygen species (ROS) are associated with the growth and invasion of malignant tumors. Ursodeoxycholic acid (UDCA) was proven to block the EGFR-MAPK signaling pathway and epithelial-mesenchymal transition (EMT)... -
- CITED
-
- Close
- SHARE
-
- Close
- Simvastatin Induces Apoptosis and Suppresses Insulin-Like Growth Factor 1 Receptor in Bile Duct Cancer Cells
-
Lee J, Hong EM, Jang JA, Park SW, Koh DH, Choi MH, Jang HJ, Kae SH
- KMID: 2363978
- Gut Liver.
- 2016 Mar;10(2):310-317.
- doi: 10.5009/gnl15195
BACKGROUND/AIMS: Statins act as antineoplastic agents through the inhibition of cell proliferation. This study sought to demonstrate the effects of statins on extrahepatic bile duct cancer cell apoptosis and to... -
- CITED
-
- Close
- SHARE
-
- Close
