- Initiation Site of Ca2+ Entry Evoked by Endoplasmic Reticulum Ca2+ Depletion in Mouse Parotid and Pancreatic Acinar Cells
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Jo H, Byun HM, Lee SI, Shin DM
- KMID: 724110
- Yonsei Med J.
- 2007 Jun;48(3):526-530.
- doi: 10.3349/ymj.2007.48.3.526
PURPOSE: In non-excitable cells, which include parotid and pancreatic acinar cells, Ca(2+) entry is triggered via a mechanism known as capacitative Ca(2+) entry, or store-operated Ca(2+) entry. This process is... -
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- Endoplasmic reticulum stress (ER-stress) by 2-deoxy-D-glucose (2DG) reduces cyclooxygenase-2 (COX-2) expression and N-glycosylation and induces a loss of COX-2 activity via a Src kinase-dependent pathway in rabbit articular chondrocytes
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Yu SM, Kim SJ
- KMID: 994401
- Exp Mol Med.
- 2010 Nov;42(11):777-786.
- doi: 10.3858/emm.2010.42.11.079
Endoplasmic reticulum (ER) stress regulates a wide range of cellular responses including apoptosis, proliferation, inflammation, and differentiation in mammalian cells. In this study, we observed the role of 2-deoxy-D-glucose (2DG)... -
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