Clin Endosc.  2024 Sep;57(5):697-699. 10.5946/ce.2024.121.

A rare cause of progressive dysphagia

• Jeon J
• Kim SE
• Oh SJ

• KMID: 2559224
• DOI: http://doi.org/10.5946/ce.2024.121

Figure

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  Fig. 1. Endoscopic findings of the esophageal lesions. (A) Not-expending esophagus. (B) Several linear, protruding lesions.

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  Fig. 2. Endoscopic findings of the stomach. (A) Diffuse wall thickening of the gastric body. (B) Hypertrophic gastric fold. (C) Relatively preserved gastric antrum.

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  Fig. 3. Histologic findings. (A) Stomach: adenocarcinoma with less glandular structure and poor cohesion that represents poorly cohesive type adenocarcinoma (hematoxylin & eosin stain, ×200). (B) Esophagus: signet ring cell infiltration that represents signet ring cell carcinoma (hematoxylin & eosin stain, ×200).

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  Fig. 4. Coronal view of F-18 fluorodeoxyglucose positron emission tomography-computed tomography. Diffuse hypermetabolic wall thickening is shown in the stomach with infiltration into the esophagogastric junction. Multiple hypermetabolic lymph nodes are shown in the perigastric, common hepatic, and abdominal para-aortic spaces. Additionally, focal lytic lesions are shown in the T9 and L4 vertebral bodies with hypermetabolism.

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