Acute Crit Care.  2021 Nov;36(4):395-396. 10.4266/acc.2021.01557.

Acute lung injury following occupational exposure to nitric acid

Affiliations
  • 1Department of Critical Care Medicine, Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea
  • 2Department of Emergency Medicine, Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea
  • 3Department of Medicine, Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea
  • 4Division of Pulmonary and Critical Care Medicine, Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea


Figure

  • Figure 1. Chest radiograph. (A) Initial chest X-ray demonstrates diffuse bilateral opacities. (B) At hospital discharge, chest X-ray showed marked improvement.

  • Figure 2. Chest computed tomography scan obtained on the day of emergency room visit presents bilateral peribronchial consolidation and ground glass opacity with sparing in the subpleural region.


Reference

1. Persinger RL, Poynter ME, Ckless K, Janssen-Heininger YM. Molecular mechanisms of nitrogen dioxide induced epithelial injury in the lung. Mol Cell Biochem. 2002; 234-235:71–80.
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2. Hajela R, Janigan DT, Landrigan PL, Boudreau SF, Sebastian S. Fatal pulmonary edema due to nitric acid fume inhalation in three pulp-mill workers. Chest. 1990; 97:487–9.
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3. Kao SL, Yap ES, Khoo SM, Lim TK, Mukhopadhyay A, Teo ST. Acute lung injury after inhalation of nitric acid. Eur J Emerg Med. 2008; 15:348–50.
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