Allergy Asthma Immunol Res.
2020 Jul;12(4):560-562. 10.4168/aair.2020.12.4.560.
Is TLR4 Critical for Neutrophil Apoptosis in Occupational Asthma?
- Affiliations
-
- 1Department of Allergy and Clinical Immunology, Ajou University School of Medicine, Suwon, Korea
- KMID: 2500738
- DOI: http://doi.org/10.4168/aair.2020.12.4.560
- Full Text Links
-
- Actions
-
Cited
- CITED
-
- Close
- Share
-
- Similar articles
-
- Toll-like Receptor 4 Deficiency Aggravates Airway Hyperresponsiveness and Inflammation by Impairing Neutrophil Apoptosis in a Toluene Diisocyanate-Induced Murine Asthma Model
- Evidence for neutrophil activation in induced sputum from patients with TDI - induced occupational asthma
- Increased neutrophil chemotactic activity is noted in alminum-induced occupational asthma
- Study of occupational asthma using an animal model
- Occupational asthma in Japan
