J Korean Soc Transplant.  2013 Jun;27(2):57-61.

Acalculous Gallbladder Perforation and Coronary Artery Aneurysm after Kidney Transplantation

Affiliations
  • 1Division of Nephrology, Department of Internal Medicine, Dongguk University Ilsan Hospital, Dongguk University College of Medicine, Goyang, Korea. kskimmd@dumc.or.kr

Abstract

Although gallbladder (GB) perforation due to acalculous cholecystitis after kidney transplantation is rarely observed, it can be life threatening and result in cholecystectomy. Coronary artery aneurysm (CAA) is also rare and may require invasive therapy depending on its diameter. We report herein the case of a 69-year-old female who developed GB perforation due to acalculous cholecystitis after kidney transplantation and underwent cholecystectomy. The patient was later invasively treated when CCA was detected by coronary angiography.

Keyword

Kidney transplantation; Acalculous cholecystitis; Coronary artery aneurysm

MeSH Terms

Acalculous Cholecystitis
Aneurysm
Cholecystectomy
Coronary Angiography
Coronary Vessels
Female
Gallbladder
Humans
Kidney
Kidney Transplantation

Figure

  • Fig. 1. Abdomen and pelvic com-puted tomography findings of the patient. (A) Cross section image revealing the irregular mucosa of gallbladder (GB) with suspicious wall defects and distended GB without definite radiopaque stone in GB. (B) Coronal section image showing a small amount of ascites in the perihepatic space and bowel ileus.

  • Fig. 2. This gallbladder (GB) specimen consists of a previously opened GB measuring 10 cm in length and 4.5 cm in diameter. The serosa is dull gray tan and the mucosa is diffusely necrotic in appearance. There is no tumorous appearance and no gall-stones, GB sludge nor GB polyps in this specimen.

  • Fig. 3. Coronary angiographic findings of the patient. (A) The short arrow indicates saccular aneurysmal change in the distal part of the obtuse marginal branch of the left circumflex artery. (B) The long arrow indicates the 3.0 mm×2.0 cm-sized tornado coil located in the aneurysmal sac. After this em-bolization, no more contrast agent filling the aneurysmal sac was observed and distal flow below aneurysmal sac disappeared.


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