- Effect of Oxygen on the Antidotal Action of Thiosulfate in Cyanide Poisoning
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Yoo KY
- KMID: 2282149
- Korean J Prev Med.
- 1982 Oct;15(1):161-166.
Cyanide poisoning is expected to be antagonized by the administration of oxygen, when it is administered in combination with the conventional cyanide antidote, sodium,thiosulfate. However, the antidotal efficacy and its... -
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- CT Brain Scan in Case of Cyanide Intoxication
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Cho PZ, Kwon KH, Sunwoo IN
- KMID: 1662063
- J Korean Neurol Assoc.
- 1986 Jun;4(1):133-136.
Eating the meat of pheasants which was killed by potassium cyanide, a 27 year old man developed several symptoms of minor intoxication. A few days later, the patient became mute,... -
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- Cyanide Poisoning Deaths Detected at the National Forensic Service Headquarters in Seoul of Korea: A Six Year Survey (2005~2010)
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Lee SK, Rhee JS, Yum HS
- KMID: 2317807
- Toxicol Res.
- 2012 Sep;28(3):195-199.
The records of 255 cyanide poisoning deaths obtained from National Forensic Service (NFS) headquarters, located in Seoul of Korea, from 2005 to 2010 were retrospectively reviewed. The mean age was... -
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- Modulation of Muscarinic K+ Channel by Protein Kinase C in Ischemic Rat Atrial Myocytes
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Kang D, Lee HS, Hong SG, Han J
- KMID: 2297959
- Korean Circ J.
- 2005 Nov;35(11):812-820.
- doi: 10.4070/kcj.2005.35.11.812
BACKGROUND AND OBJECTIVES: Recent studies have shown that many kinds of K+ channels, including the muscarinic K+ channel (KACh), are activated in the ischemic heart. It is known that these... -
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- Inhibiting Cytochrome C Oxidase Leads to Alleviated Ischemia Reperfusion Injury
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Yang Z, Duan Z, Yu T, Xu J, Liu L
- KMID: 2377459
- Korean Circ J.
- 2017 Mar;47(2):193-200.
- doi: 10.4070/kcj.2016.0137
BACKGROUND AND OBJECTIVES: The overall purpose of this study was to investigate the role of cytochrome C oxidase (CcO) in preventing ischemia reperfusion-induced cardiac injury through gaseous signaling molecule pathways. MATERIALS... -
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- Alterations in Membrane Transport Function and Cell Viability Induced by ATP Depletion in Primary Cultured Rabbit Renal Proximal Tubular Cells
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Lee SJ, Kwon CH, Kim YK
- KMID: 2285366
- Korean J Physiol Pharmacol.
- 2009 Feb;13(1):15-22.
- doi: 10.4196/kjpp.2009.13.1.15
This study was undertaken to elucidate the underlying mechanisms of ATP depletion-induced membrane transport dysfunction and cell death in renal proximal tubular cells. ATP depletion was induced by incubating cells... -
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